In a study intended to depict the role of antiapoptotic protein Mcl-1 in resisting DNA damage induced by chemotherapy in prostate cancer, BA combined with ENMD-1198, an antitumor agent, significantly increased apoptotic/necrotic cell death and inhibited metastasis by decreasing Mcl-1, which led to increased DNA damage [72]. Here, MCL1 is linked to prostate carcinoma.