Present data revealed that BPA treatment did not affect the expression rate of maintaining and de novo DNA methylation enzymes (i.e., Dnmt1 and Dnmt3a), which are notably involved in the BPA dependent occurrence of insulin resistance, hepatic lipid accumulation, and steatosis in adulthood via aberrant methylation status in the promoter region of genes involved in glucose and lipid homeostasis [25,65,66]. Here, DNMT3A is linked to steatosis.