Given the short half-life (24–36 h) of sBCMA compared to IgG or IgA immunoglobulins (21 or 7 days, respectively), BCMA might be not only a biomarker for the diagnosis of MM, but also for monitoring disease progression and treatment response, even in non-secretory MM patients, for whom no accurate monitoring for therapy efficacy has been available [2]. Here, CD79A is linked to Miyoshi myopathy.