Previous studies have evaluated the molecular features of these neoplasms, focusing mainly on the molecular pathways related to the maintenance of telomere length, as well as to various genes involved in cell survival or apoptosis or in the control of invasiveness (SMO, TRAF7, AKT1, KLF4, and SMARCB1) [30,31,32]. The gene discussed is SMARCB1; the disease is neoplasm.