Effectively, the study focused on SREBP-1c involvement in the same rat model of NAFLD [14] showed that the dysregulation of triglycerides and free fatty acids (FFAs), leading to hepatic steatosis and induction of oxidative and inflammatory stress, favoring the progression of NAFLD, were markedly decreased in rats fed an HF-F, DHEA-fortified diet [14]. This evidence concerns the gene SREBF1 and hydrops fetalis.