AKT1 and esophageal cancer: Similar results were obtained in esophageal cancer cells, after inhibition of miR-200c, which saw an increase in apoptosis, sensitivity to cisplatin, and PPP2R1B expression, which encodes the β-isoform of the A subunit of another serine/threonine phosphatase PP2A, followed by a reduction of p-AKT [34].