Since from a neuropathological point of view, both PD and AD are proteinopathies with a long prodromal period characterized by hyposmia [48], it could be suggested that UA may also show potential against impaired olfaction caused by beta-amyloid deposition and a neurofibrillary tangle of the tau formation in the olfactory tract. The gene discussed is MAPT; the disease is Alzheimer disease.