Interestingly, monocytes from acute COVID-19 showed a predominant upregulation of anti-inflammatory genes, such as IL-10, BCL6, MAFF, MAFG and AREG, together with downregulation of pro-inflammatory cytokines such as TNF-α and CCL3 and downregulation of the antigen presentation molecule HLA-DRA (in agreement with flow cytometry data) or CD4. The gene discussed is CCL3; the disease is COVID-19.