Re-analysis of the TCGA PRAD cohort data (n = 623) showed that regardless of sample type, the expression of AIM/CD5L and PSA/KLK3 is inversely correlated (Figure 1C), such that relative to AIM, PSA transcript expression was significantly overexpressed (7.41-fold, p < 0.0001) (Figure 1D). The gene discussed is KLK3; the disease is prostate adenocarcinoma.