The experimental studies in postnatal exposure (prepuberal age) to physical immobilization, maternal separation, or transportation stress models showed significantly lower values of kidney weight, and glomerular volume density in the stressed group [31,32], as well as sensitization to arterial hypertension after angiotensin II infusion [33], exaggerated gene expression immune response (IL-1β and T cells) [36], reduced alfa-adrenergic receptor (α-AR) density in renal vasculature [29], and cardiorenal metabolic alterations [30]. The gene discussed is IL1B; the disease is Hypertension.