VAV3 and hypertensive disorder: The migration of axons of GABAergic neurons located in the CVLM toward their target neurons of the RVLM is impaired in the absence of Vav3 (Figure 3, point a) [54], leading to the unleashing of RVLM activity, the hyperactivation of the SNS, and the development of SNS-dependent defects such as hypertension, tachypnea, and hypercapnia (Figure 3, point a) [54,60].