Among them, B cells, which express GARP when activated by TLR ligands such as TLR4, TLR7 and TLR9, have been only explored in the context of autoimmune disease [12] and hepatic stellate cells, for which GARP is required to anchor and activate LTGF-β, are currently being explored in liver fibrosis [16]. This evidence concerns the gene LRRC32 and autoimmune disease.