As shown in our data, ox-LDL–treated HTR8/SVneo cells exhibited preeclampsia-like phenotypic changes: declined migration (Figure 3C), impaired angiogenesis (VEGFA was downregulated and FLT1 upregulated), and apoptosis (Bax was upregulated) (Figure 3A), which were in accordance with the expression in the PE placenta. The gene discussed is BAX; the disease is preeclampsia.