CYP19A1 and hereditary elliptocytosis: Furthermore, isothiocyanato-cyclohexane was characterised as a common environmental pollutant and its increase in HE patients was attributed to impaired liver catabolism, whereas increased 1-methyl-4-(1-methylethenyl)-benzene levels again in HE patients may have originated from an enhanced aromatase activity due to extensive alcohol abuse that could have been responsible for changes in metabolism.