Age is a primary risk factor for the development of symptoms (although HD is always caused by a dominant autosomal mutation in the huntingtin gene; the development of symptomology generally occurs with older age), cerebral protein deposition is present in each condition (α-synuclein in PD, tau and amyloid-β in ADD, and huntingtin in HD), and the clinical presentation can show significant overlap, with increased risk of psychological complaints [7], issues with sleep [8], and difficulty walking [9], as well as the progressive development of cognitive impairment [10,11]. This evidence concerns the gene MAPT and Huntington disease.