H2AX and cervical carcinoma: Moreover, according to published data, miR‐24 regulates the expression of tumor suppressor/senescence‐associated proteins differently depending on the cell type and metabolic state of the cell: It reduced p16 protein levels in human diploid fibroblasts and cervical carcinoma cells or highly senescent cells (Figure S3), and it inhibited H2Ax in terminally differentiated hematopoietic cells making them vulnerable to DNA damage (Lal et al., 2009).