In NSCLC, highly expressed miR‐802 was found to be able to target to inhibit the expression of FGFR1, p‐PI3K, p‐AKT, and p‐mTOR, thereby inhibiting the proliferation, invasion, metastasis, and colony formation of HCC cells, and inducing apoptosis of HCC cells.34 This evidence concerns the gene AKT1 and non-small cell lung carcinoma.