The neutrophil engulfment by dermal macrophages is mediated by the expression of tyrosinase kinases receptors, such as Axl and MertK, and has been recently shown during in vivo infection with L. major, where an up-regulation of wound-healing phenotype markers, such as resistin-like molecule alpha (Relmα) and Arg1 was reported (135). The gene discussed is ARG1; the disease is infection.