It was previously shown that infection with the porcine transmissible gastroenteritis virus (TGEV) belonging to the coronavirus family can cause an increase of FcRn expression in the normal porcine intestinal cell line IPEC-J2 by activating NF-κB signaling through membrane receptors of the Toll-like receptor family and increasing pro-inflammatory cytokine levels (157, 158). Here, NFKB1 is linked to infection.