CD36 appears to play an active role in the pathogenesis ofMycobacterium bovisBacillus Calmette-Guérin infection, with CD36−/−mice exhibiting decreased mycobacterial burden and lower numbers of hepatic granulomas, and CD36−/−macrophages restricting mycobacterial growth in vitro.53In addition, CD68 on Kupffer cells acts as a putative receptor forPlasmodium bergheisporozoites,54playing a significant role in the hepatic invasion stage of the malaria-causing parasite's lifecycle. Here, CD36 is linked to malaria.