SOAT1 and staphylococcus aureus infection: The up-regulated DEGs were mainly related to osteoclast differentiation, chemokine signaling pathways, natural killer cell-mediated cytotoxicity, cytokine-cytokine receptor interactions, phagosomes, the JAK-STAT signaling pathway, tuberculosis, the Fc epsilon RI signaling pathway, Staphylococcus aureus infection, and complement and coagulation cascade pathways (Figure 3C).