Considering first all FOXC1 and RUNX1 binding sites in Fujioka AML cells, we found 5,246 genomic locations where the absolute summit of a FOXC1 peak was 200 bp or closer to the absolute summit of a RUNX1 peak (i.e., 28.0% of FOXC1 peaks and 15.3% of RUNX1 peaks) (Figures S4A and S4B). The gene discussed is RUNX1; the disease is acute myeloid leukemia.