Earlier we reported that in the tears of AKC patients, as in patients with SJS, but not in healthy controls, interleukin (IL)−6, IL‐8, eotaxin, and macrophage inflammatory protein−1b were significantly upregulated and that the level of total IgE was significantly higher in AKC patients than in patients with SJS and in the healthy controls.8 This evidence concerns the gene CCL11 and Schwartz-Jampel syndrome.