Wu et al. in their study showed that in ankylosing spondylitis as an autoimmune disease, the structural constituent of the ribosome, SRP-dependent cotranslational protein targeting to membrane, nonsense-mediated decay, translation, viral transcription, nuclear-transcribed mRNA catabolic process, poly(A) RNA binding, and translational initiation term were enriched [58]. Here, UCN2 is linked to autoimmune disease.