Additional evidence about the role of the Th17/IL-17 axis on Kidney diseases comes from the observation of the increased risk of CKD associated with renal inflammation in human diseases that occur with the hyperactivation of the Th17/IL-17 axis, like psoriasis (210–212), rheumatoid arthritis (213, 214), and ankylosing spondylitis (215–217). This evidence concerns the gene IL17A and rheumatoid arthritis.