We used a human cervical cancer cell line, HeLa, as an ideal reconstitution system since it lacks endogenous expression of RIPK3.[16] In the dark, both mCherry (mCh)‐CRY2‐RIPK1 and RIPK3‐GFP exhibited an even distribution in the cytosol of transfected HeLa cells. This evidence concerns the gene RIPK3 and cervical cancer.