SOAT1 and viral infectious disease: On the basis of the network pharmacology analysis, GJHQHLRSD achieves anti-inflammatory effects by regulating 141 pathways, included inflammation as well as cancer-related and viral infection signaling pathways, such as the “EGFR signaling pathway,” “MicroRNAs in cancer,” “TNF signaling pathway,” and “JAK-STAT signaling pathway.” Although these pathways have been shown to be strongly associated with UC, some pathways have not yet been shown to be regulated by GJHQHLRSD.