CCR2 and infection: However, despite the fact that no CCR2+ inflammatory monocytes were detected in the spleen of CCR2 knockout mice (Supplementary Fig. S9D), these mice had similar peak parasitaemias as their wild-type C57Bl/6J mice for both infections initiated with RMT and SBP P. chabaudi [p = 0.9486 and p = 0.2990 respectively; Fig. 6E–F and (Sponaas et al., 2009)], indicating that bone marrow-derived CCR2+ inflammatory monocytes are not involved in attenuation of infection.