We next made use of mice in which the IFNγR2 chain, essential for transducing the IFNγ signal (Hemmi et al., 1994; Lee et al., 2015), was selectively deleted on two different populations of myeloid cells: LysMcre+/-Ifnγr2fl/fl (granulocytes, monocytes and macrophages), and CD11ccre+/-Ifnγr2fl/fl (classical and plasmacytoid dendritic cells, some B cells); and compared the infections with those in mice in which IFNγ-signalling in CD4+ and CD8+ T cells (CD4cre+/-Ifngr2fl/fl) or B cells (CD19cre+/-Ifnγr2fl/fl) was deleted. The gene discussed is IFNG; the disease is infection.