CCR2 and infection: Although there was a clear CCR2-dependent influx of bone-marrow-derived inflammatory monocytes in RMT P. chabaudi infections similar to that observed previously in SBP infections (Sponaas et al., 2009), which may have iRBC phagocytic activity, we ruled out a role for these cells as lack of CCR2 in knockout mice had no impact on the magnitude or duration of an acute RMT infection.