After myocardial infarction, a cytokine storm induces myofibroblast differentiation via angiotensin II and TGFβ, among others (Frangogiannis, 2015), Myofibroblasts have a greater contractile and invasion capacity and also a higher production of extracellular matrix components; hence, the excessive differentiation to myofibroblasts results in scar formation with no contraction capacity. Here, AGT is linked to myocardial infarction.