The common biological pathways and its interaction with the inflammatory factors need to be established to uncover the pathogenesis of SJS. Our study shows similar trends with respect to expression pattern of inflammatory cytokines studied previously in chronic SJS patients but differ with the level of significance of IL-10, IL-15, IL-17A, Bfgf, RANTES/CCL5, MCP-1, GMCSF, MIP1B/CCL4, TNF-α, and IFN-γ12. The gene discussed is TNF; the disease is Schwartz-Jampel syndrome.