NLRP3 and intracranial hemorrhage: On the one hand, the activation of NLRP3 inflammasomes produces high levels of inflammatory cytokines, recruits other immune cells, and induces an inflammatory response to eliminate the risk-related molecular pattern protein (DAMP) after hemorrhagic stroke; On the other hand, the excessive activation of NLRP3 inflammasomes can lead to persistent inflammation and brain damage after intracranial hemorrhage.