PRC1 and cervical squamous intraepithelial neoplasia: Pharmacologic CIN induction led to an expected increase in chromosome missegregation and micronuclei formation (Supplementary Fig. 11a, b), as well as transcriptional changes accounting for half of genes (50.4%) that were differentially expressed upon PRC1 inhibition (Supplementary Fig. 11c), including upregulation of inflammatory and migratory pathways (Supplementary Fig. 11d).