Expanding on these earlier findings, Nystrom’s group deployed multiple methods of analysis to unveil provocative age‐ and site‐dependent differences in fibrosis in this same murine model of RDEB, association between inflammation and fibrosis progression, and promising reductions in fibrosis with the use of Ang‐(1‐7) (Bernasconi et al, 2021). Here, ANG is linked to recessive dystrophic epidermolysis bullosa.