Minor populations of cardiac endothelial cells express PDGFRα with an increase in CD31+PDGFRα+ cells seen post-MI, indicating that the PDGFRα is conducive to post-MI angiogenic regulation during cardiac wound healing (Zhao et al., 2011; Awada et al., 2015; Farbehi et al., 2019). The gene discussed is PDGFRA; the disease is myocardial infarction.