Importantly, to determine the role of viral replication in the control of STAT expression, infections were performed either in the presence or in the absence of the DNA replication inhibitor cytosine arabinoside (AraC), which is very well known to block the viral cycle prior to viral DNA replication, and to prevent late ASFV genes expression (Rodríguez and Salas, 2013). This evidence concerns the gene SOAT1 and infection.