Moreover, sulfasalazine (an anti-inflammatory drug) has shown inhibitory action of NF-κB that triggers the apoptosis in glioma cells (Robe et al., 2004), and a decoy oligonucleotide strategy to prevent NF-κB that played a crucial role in the decline in the number of cells (Gill et al., 2002). This evidence concerns the gene NFKB1 and glioma.