A study to investigate the underlying mechanism of NEAT1 in DLBCL progression found that NEAT1 transcriptionally regulated by MYC was upregulated in DLBCL tissues and cell lines, and NEAT1 knockdown resulted in the inhibition of DLBCL cell proliferation and a promotion of DLBCL cell apoptosis. Here, MYC is linked to diffuse large B-cell lymphoma.