They observed that NEAT1 was upregulated in glioma tissues and cell lines, and this upregulation contributed to glioma progression by inducing glioma cell survival, promoting cell proliferation, migration, and invasion by sponging miR-107 118, miR-132 119, and miR-449b-5 120 to elevate the expression levels of cyclin-dependent kinase 14 (CDK14), SRY-box transcription factor 2 (SOX2), and c-Met, respectively. The gene discussed is MET; the disease is glioma.