It was shown, indeed, that BCR/Abl-positive cells undergo intense glycolysis and glutaminolysis [33], that glycolytic cancer cells in general consume more glutamine than their normal counterparts [34] and that the amount of glucose converted to lactate is increased when murine K-Ras-transformed fibroblasts are grown in the presence of glutamine [35]. The gene discussed is ABL1; the disease is cancer.