The expression of miR-4802 and miR-18a was indirectly repressed by Fusobacterium (F.)nucleatum, a component of the gut microbiota highly represented in drug-resistant colon cancer patients, resulting in the upregulation of autophagy-related 7 (ATG7) and unc-51-like autophagy activating kinase 1 (ULK1) targets, two activators of autophagy, as well as a resistance to 5-FU and L-OHP [64]. Here, ATG7 is linked to colonic neoplasm.