A recent study showed that a combination of interleukin-13 (IL-13), granulocyte-colony-stimulating factor (G-CSF), and granulocyte-macrophage-colony-stimulating factor (GM-CSF) induced CD11b+Gr1+ cells that migrated to allo-priming sites and robustly suppressed graft-versus-host disease when administered in animals [25]. The gene discussed is IL13; the disease is graft versus host disease.