Therefore, the key mechanism of M. alba L. leaves against gout might be to suppress the inflammasomes in synovial fluids by inhibiting AKT1 by γ-Tocopherol, PRKCA by 4-Dehydroxy-N-(4,5-methylenedioxy-2-nitrobenzylidene) tyramine, and PLA2G2A by Lanosterol acetate on the RAS signaling pathway (Figure 10). The gene discussed is PRKCA; the disease is gout.