EPHA2 and infection: EphA2, where Eph stands for erythropoietin-producing human hepatocellular carcinoma cell line, was identified as HHV-8 entry receptor by Hahn and colleagues who showed that deletion of the EphA2 gene abolished infection of endothelial cells and that binding of gH/gL to EphA2 on cells led to increased EphA2 phosphorylation and endocytosis facilitating viral entry [10].