As the cells were also CCR9+, integrin α4β7+, and CXCR3+, these data would be compatible with the notion that there is some CXCR5‐dependent T‐B cell interaction in CeD, possibly occurring in germinal centers of secondary or tertiary lymphoid structures in the intestine. The gene discussed is CXCR3; the disease is cranioectodermal dysplasia.