Indeed, we have previously demonstrated that that the number of gluten‐specific cells increases in the small intestine with oral gluten challenge,[7] and by global RNA analysis of gut biopsies it was demonstrated that CXCR6 is increased in CeD patients compared to controls.[33] The CXCR6/CXCL16 axis is important to the recruitment of tissue‐resident CD8+ memory T cells in the airways,[34] and in mouse models, to the positioning of disease‐specific T cells within the inflamed tissue.[35, 36] Notably, CXCR6 was not included in the panel staining CD8+ and γδ+ T cells. This evidence concerns the gene CD8A and cranioectodermal dysplasia.