IFNA1 and COVID-19: Several studies showed that SARS-CoV-2, compared to other viruses, boosts the production of inflammatory mediators while delaying and/or dampening antiviral IFN responses in patients with severe COVID-19 (Blanco-Melo et al., 2020; Galani et al., 2021; Hadjadj et al., 2020; Mudd et al., 2020).