These findings were followed up by a series of association studies by Lal et al. that targeted exon-disrupting microdeletions in RBFOX1, revealing a significant excess among GGE patients compared to population controls, (Lal et al., 2013b) as well as among cases of Rolandic epilepsy (Lal et al., 2013a) and sporadic focal epilepsy, (Lal et al., 2015) suggesting that RBFOX1 deletions are involved in a wide spectrum of both focal and generalized epilepsies. Here, RBFOX1 is linked to idiopathic generalized epilepsy.