Besides, TRAIL-coated gold nanoparticles (TRAIL-AuNPs) robustly induced apoptosis in NSCLC by inducing mitochondrial fragmentation in tumor cells along with a marked promotion in mitochondrial recruitment of dynamin-related protein 1 (Drp1), inducing mitochondrial deficits, and supporting the autophagy process (172). This evidence concerns the gene DNM1L and neoplasm.