Patients suffering from SARS have respiratory complications (Li and Ma, 2020) that are related to the ability of SARS-CoV-2 to penetrate endothelial cells through a mechanism that involves angiotensin-converting enzyme-2 (ACE-2) receptors located at the luminal surface of the lungs (Le Bert et al., 2020; Li and Ma, 2020) as shown in Figure 1. This evidence concerns the gene ACE2 and severe acute respiratory syndrome.