Several studies had showed that Myosin 1 C (MYO1C) may play a key role in regulatingautophagosome–lysosome fusion through F-actin remodeling, and miR-137 overexpression inhibited the cell migration, proliferation by targeting Krűppel-likefactor 12 (KLF12) and MYO1C in gastric cancer cell lines [58,59]. This evidence concerns the gene MYO1C and gastric cancer.