In our experiments, knock down of AKT1 and AKT2 completely reverted Th17‐induced resistance in AKT3‐negative cervical cancer cell lines SiHa and HeLa and also in the AKT3 expressing cell line SW756, indicating that the more ubiquitously expressed isoforms AKT1 and AKT2 were sufficient for Th17‐induced resistance. The gene discussed is AKT3; the disease is cervical cancer.