A recent investigation demonstrated that circBACH1 was significantly upregulated in HCC tissues and interacted with HuR (an extensively studied RBP that can inhibit p27 translation via an interferon-responsive sequence element in the p27 5′-untranslated region) to promote HuR translocation and facilitate its accumulation in the cytoplasm, thereby downregulating p27 expression [48, 72, 73]. Here, ELAVL1 is linked to hepatocellular carcinoma.